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Ibutamoren

  • Hormone
  • Still being tested
  • Identity checked
  • Sources last checked 2026-09-04

This page shows what was measured, who it was measured in, and what that does not settle.

What Ibutamoren does in the body

Age-related muscle loss and fracture recovery — investigational, and the programme ended

Ghrelin is the hormone the stomach releases when it is empty; it makes you hungry and it tells the pituitary to release growth hormone. Ibutamoren copies ghrelin at its receptor, so it does both: appetite rises and growth hormone comes out in the pulsatile pattern of a young adult, which raises IGF-1 in the blood. That part is not in dispute and never has been. What Merck tested, repeatedly and expensively, was whether that translated into people being stronger, recovering better or thinking more clearly. In three different populations the answer was no.

What happened in people

Fat-free mass +1.1 kg against -0.5 kg on placebo over one year, confirmed by two-year exploratory analysis

Source-linked record

Where this came from

A person wrote this into the record with the study named beside it. No reviewer has signed it off.

The recorded finding that sits closest to something a person would notice. Written into the record, not signed off, and it carries no population or interval.

The limit that matters most

That raising IGF-1 produces functional benefit — tested in three populations across 912 randomised participants and supported in none

Where it acts
Growth hormone secretagogue receptor on anterior pituitary somatotrophs and in the hypothalamic arcuate nucleus
Kind of result
A number that stands in for health
Supervision
Professional supervision is normally required. It is a prescription or clinician-administered medicine where it is approved.

What the registries record it as

  • The substance registry classes this as chemical.

    FDA substance registry · GJ0EGN38UL · read 2026-08-29

  • The supplement label database classes it as other, under the name Ibutamoren.

    NIH Dietary Supplement Label Database · 3997 · read 2026-08-29

  • Its recorded molecular formula is C27H36N4O5S, weighing 528.7.

    PubChem record · 178024 · read 2026-08-29

Where each sentence above came from

No recorded sentence describes the change this makes in a way that stands on its own. The page opens with what it is taken for instead, and the recorded explanation follows below.

Shown as the opening line on this page.

A limit recorded against this substance. Not signed off as a reviewed claim.

The four opening statements run to 125 words.

Words this page uses

Four words worth knowing first

Chosen from what this page shows, with each one explained before the word it depends on.

Comparator

A comparator is whatever the treatment was measured against.

A picture of it, and where the picture fails

It is like the other runner in a race.

Where that stops being true. A race has one winner. A study can show both arms improved.

What people get wrong. Results are read without asking what the other group got. Beating nothing is not beating a treatment.

The control condition against which the experimental intervention is assessed.

Randomisation

Randomisation means chance decides who gets which treatment.

A picture of it, and where the picture fails

It is like a coin toss deciding the groups.

Where that stops being true. A coin toss is fair once. Fairness here comes from doing it for everyone.

What people get wrong. It is thought to make groups identical. It makes them similar on average, including on things nobody measured.

Allocation of participants to arms by a chance process, so that unmeasured factors are balanced in expectation.

Absolute difference

An absolute difference is how many more people in a hundred were affected.

A picture of it, and where the picture fails

It is like counting heads in two rooms of a hundred.

Where that stops being true. Heads are easy to count. Study results carry a margin of error too.

What people get wrong. It is confused with a percentage change, which can look far larger.

The arithmetic difference in event rates between arms.

Confidence interval

A confidence interval is the range the true answer is likely to sit in.

A picture of it, and where the picture fails

It is like a weather forecast giving a range rather than one number.

Where that stops being true. A forecast range covers tomorrow. This one covers what the study could resolve.

What people get wrong. The middle number is read as the answer. A range crossing zero means no change is still possible.

An interval estimate that would contain the true parameter in a stated proportion of repeated studies.

What happened in peopleRead from sources, not yet reviewed

What happened in people

Each card is one study: the exact question it asked, who was in it, and whether it showed what it set out to show.

Fat-free mass and abdominal visceral fat after 1 year

The study showed what it set out to show

Who was studied
Nass 2008 two-year trial in healthy older adults
How many people
65
Study design
Phase 2, randomised, double-blind, placebo-controlled, modified crossover
Compared against
A dummy treatment
Kind of result
Measured performance
What was found
Fat-free mass +1.1 kg (95% CI 0.7 to 1.5) versus -0.5 kg (-1.1 to 0.2), P < 0.001; no significant difference in abdominal visceral fat
Repeated elsewhere
Unreplicated

What this does not prove. Meeting one endpoint in one population does not carry to other goals or other people.

The limits of this study, and where it came from

Main limit. Fasting glucose +0.3 mmol/L (P = 0.015) with decreased insulin sensitivity; cortisol +47 nmol/L (P = 0.020); increased appetite, transient lower-extremity oedema and muscle pain. Increased fat-free mass did not produce any change in strength or function.

How far it carries. This study is recorded from the curated record, not from the registry match.

Form and route. Oral tablet, capsule or liquid, 25 mg once daily in every published trial

Interval reported. 95% CI 0

Written into the record, not signed off as a reviewed claim.

Mean change from baseline at month 12 on CIBIC-plus, ADAS-Cog, ADCS-ADL and CDR sum of boxes

The study did not show it

Who was studied
Sevigny 2008, MK-677 Protocol 30 in Alzheimer disease
How many people
563
Study design
Randomised, double-blind, placebo-controlled, multicentre, 12 months
Compared against
A dummy treatment
Kind of result
A number that stands in for health
What was found
No significant differences between groups on any measure, despite IGF-1 rising 60.1% at 6 weeks and 72.9% at 12 months
Repeated elsewhere
Unreplicated

What this does not prove. A study that did not show an effect does not show that no effect exists anywhere.

The limits of this study, and where it came from

Main limit. No limitation is recorded for this study.

How far it carries. This study is recorded from the curated record, not from the registry match.

Form and route. Oral tablet, capsule or liquid, 25 mg once daily in every published trial

Interval reported. Not recorded in this summary

Written into the record, not signed off as a reviewed claim.

Rank analysis of change in objective functional performance measures and in blood IGF-1

The study did not show it

Who was studied
Adunsky 2011 phase 2b in hip fracture recovery
How many people
123
Study design
Phase 2b, randomised, double-blind, placebo-controlled, multicentre
Compared against
A dummy treatment
Kind of result
What a body can do day to day
What was found
Stair climbing power +12.5 W (95% CI -10.95 to 35.88, P = 0.292); gait speed 0.7-score difference (95% CI 0.17 to 1.28, P = 0.011); IGF-1 +51.4 ng/mL (P < 0.001); no improvement in several other functional measures
Repeated elsewhere
Failed to Replicate

What this does not prove. A study that did not show an effect does not show that no effect exists anywhere.

The limits of this study, and where it came from

Main limit. The trial was terminated early because of a safety signal of congestive heart failure in a limited number of patients. The authors conclude the compound has an unfavourable safety profile in this population.

How far it carries. This study is recorded from the curated record, not from the registry match.

Form and route. Oral tablet, capsule or liquid, 25 mg once daily in every published trial

Interval reported. 95% CI -10

Written into the record, not signed off as a reviewed claim.

Change from week 6 to week 26 in a panel of functional performance measures

The study did not show it

Who was studied
Bach 2004 hip fracture trial across 13 centres
How many people
161
Study design
Randomised, double-blind, placebo-controlled, 6 months treatment plus 6 months follow-up
Compared against
A dummy treatment
Kind of result
What a body can do day to day
What was found
IGF-I rose 84% (95% CI 63 to 107) versus 17% (8 to 28) on placebo; no significant differences in functional performance measures or overall Sickness Impact Profile score
Repeated elsewhere
Replicated

What this does not prove. A study that did not show an effect does not show that no effect exists anywhere.

The limits of this study, and where it came from

Main limit. No limitation is recorded for this study.

How far it carries. This study is recorded from the curated record, not from the registry match.

Form and route. Oral tablet, capsule or liquid, 25 mg once daily in every published trial

Interval reported. 95% CI 63 to 107) versus 17% (8 to 28) on placebo; no significant differences in functional performance measures or overall Sickness Impact Profile score

Written into the record, not signed off as a reviewed claim.

What we know
RNAWiki holds 4 written-up human studies for this substance, each with the question it asked.
How we know it
Each card names the study, the number of people and what the study set out to measure.
What this does not prove
These summaries were written by a person and have not been signed off as reviewed claims.
Why it matters
A study that failed is as informative as one that worked, and both are here.
What we still do not know
No reviewed claim exists, so no exact population, effect size or interval is published yet.

What happened in peopleRead from sources, not yet reviewed

How close this is to real life

The top step is something a person would feel or care about. The bottom is a guess from software. Filled steps are where evidence exists for this substance.

  1. Living longer, or avoiding a major event No evidence recorded. Death, a heart attack, a stroke, a hospital stay.No registered study measures this.
  2. What a body can do day to day No evidence recorded. Walking, dressing, breathing, recovering.No registered study measures this.
  3. Measured performance No evidence recorded. How much was lifted, how far was run, how fast.No registered study measures this.
  4. Symptoms and quality of life No evidence recorded. Pain, tiredness, mood, sleep, as the person rated it.No registered study measures this.
  5. A number that stands in for health No evidence recorded. Cholesterol, blood sugar, bone density. A stand-in, not the thing itself.No registered study measures this.
  6. A step measured inside a person Evidence recorded. Something measured in human tissue or human cells.A human record sits on the stored organism ladder.
  7. Animals No evidence recorded. Mice, rats, dogs. Useful for ideas, not proof about people.No animal record is stored.
  8. Cells in a dish Evidence recorded. Cells or chemistry on a bench, far from a whole body.Stored mechanism abstracts describe steps measured in cells or tissue.
  9. A guess from software No evidence recorded. Nobody measured it. RNAWiki never publishes one as a finding.RNAWiki stores no prediction for this record. Software can suggest a link. RNAWiki does not publish one as a finding.

Higher on these steps means closer to something a person would feel. It does not mean better done.

What it changes in the bodyRead from sources, not yet reviewed

The path through the body

From what a person takes to what changes. A solid line is a step measured in people. A dashed line is a step nobody has measured that way.

  1. Start

    Ibutamoren

    What a person takes: Oral tablet, capsule or liquid, 25 mg once daily in every published trial.

    The measurement behind this step

    A once-daily oral dose. The unusual consistency of the clinical programme — 25 mg in all four major trials — means the human dose-response is narrow but the exposure is well characterised at that dose, over durations up to two years. That is far more human data than any SARM on this site has.

  2. Getting in

    Swallowed once daily at 25 mg

    An oral tablet or capsule. Every published trial used the same dose, 25 mg once a day.

    Described, with no measurement named. No measurement is named for this step, so it is drawn as an unconfirmed link.

    The measurement behind this step

    Orally active non-peptide spiroindane. Twenty-five milligrams once daily is the dose in Bach 2004, Nass 2008, Sevigny 2008 and Adunsky 2011 alike, which is unusual and makes the trials directly comparable.

  3. Reaching the cell

    Reaches the pituitary and the hypothalamus

    It crosses into the brain regions that control growth hormone release and appetite.

    Described, with no measurement named. No measurement is named for this step, so it is drawn as an unconfirmed link.

    The measurement behind this step
  4. What it acts on

    Activates the ghrelin receptor

    It binds the receptor that the hunger hormone ghrelin normally uses, and switches it on.

    Described, with no measurement named. No measurement is named for this step, so it is drawn as an unconfirmed link.

    The measurement behind this step

    Agonist at GHS-R1a, a Gq-coupled receptor. Activation raises intracellular calcium in somatotrophs and, in the arcuate nucleus, drives NPY and AgRP neurons — which is why appetite increase is the commonest adverse effect and was reported in the trials as subsiding over a few months.

  5. The change it makes

    Growth hormone pulses rise, and IGF-1 follows

    The pituitary releases growth hormone in bursts, more of them and bigger, and the liver responds by making more IGF-1.

    Described, with no measurement named. No measurement is named for this step, so it is drawn as an unconfirmed link.

    The measurement behind this step

    Enhanced pulsatile GH secretion rather than a continuous elevation, preserving the physiological pattern. Hepatic GH receptor signalling then raises circulating IGF-1 by 50% to 85% depending on the population, an effect that was measured and confirmed in every trial of the compound.

  6. What that does for a person

    Fat-free mass rises; the clinical outcomes did not

    Scales and scans show more lean tissue and more body weight. Strength tests, walking tests, recovery from fracture and cognitive scores did not move.

    Measured in people. Written by a person from the study named beside the step. Not signed off as a reviewed claim.

    The measurement behind this step

    Fat-free mass +1.1 kg against -0.5 kg on placebo over one year, confirmed at two. No change in strength or function in the same trial; no significant functional benefit in the larger hip fracture trial; no effect on four Alzheimer disease scales in 563 patients. Fasting glucose rose and insulin sensitivity fell, the expected metabolic consequence of sustained growth hormone elevation.

No suggested links are held for this record, so nothing is hidden from this path.

What we know
The record describes 5 steps from taking it to an effect in a person.
How we know it
Each step names the study behind it in the technical detail beside it.
What this does not prove
A change inside the body is a reason to look. It is not a result in a person.
Why it matters
A reader can see exactly where the chain stops being measured in people.
What we still do not know
No reviewed claim binds any of these steps to a named population.

What is missing or unclearRead from sources, not yet reviewed

Were people like you studied?

RNAWiki cannot tell whether a study fits you. It can show who was in it, and where the result stops carrying.

Who was studied

People similar to this profile were included.

  • In trials: 65 healthy adults aged 60 to 81 over two years, 161 and then 123 patients recovering from hip fracture, 563 patients with mild to moderate Alzheimer disease, and postmenopausal women with osteoporosis. Outside trials: people wanting appetite, sleep and muscle gains, mostly alongside SARMs.

Who is missing from the studies

  • Which groups were under-represented in the studies has not been recorded for this substance. Pregnancy, breastfeeding, children, older adults, and people with liver or kidney conditions are commonly excluded from trials; nothing here says whether they were.

Where the result stopped carrying

  • No effect on any of four clinical scales in 563 patients with Alzheimer disease despite unambiguous target engagement
  • No significant functional benefit in either hip fracture trial, and the second was terminated early for a congestive heart failure signal
  • No change in strength or function in the two-year healthy-ageing trial, which the authors state explicitly
  • This is a scope explorer, not a diagnosis engine.
  • It shows who was studied so you can see whether people similar to you were included.

RNAWiki cannot tell whether a study fits you. It can show who was in it.

What it would be like to takeRead from sources, not yet reviewed

Why it might seem to do nothing

A real effect and a noticed effect are different things. These are the recorded reasons the two come apart.

It was studied in different people

The people in the studies were not much like the reader.

On this record: Which groups were under-represented in the studies has not been recorded for this substance. Pregnancy, breastfeeding, children, older adults, and people with liver or kidney conditions are commonly excluded from trials; nothing here says whether they were.

The evidence may simply be wrong

Small early studies often do not hold up.

On this record: RNAWiki has not yet published a reviewed conclusion for this use.

Other reasons RNAWiki checked and found nothing for (11)
It was studied for a different goal
The studies measured something else entirely. Nothing in this record points to this reason.
A different form was studied
The studied form is not the form on the shelf. Nothing in this record points to this reason.
There was nothing to correct
Where a level is already normal, topping it up may change nothing. Nothing in this record points to this reason.
Something else had to happen too
In the studies it was paired with training, a diet or another treatment. Nothing in this record points to this reason.
The change is too small to feel
A real change can still sit below what a person notices. Nothing in this record points to this reason.
Day-to-day swing hides it
Sleep, food, stress and time of day move most numbers more than this would. Nothing in this record points to this reason.
Not enough time yet
The studies ran longer than the person has waited. Nothing in this record points to this reason.
It was not taken as studied
Missed days change the result, and studies count them. Nothing in this record points to this reason.
Something else changed at the same time
Two changes at once cannot be told apart afterwards. Nothing in this record points to this reason.
The product may not be what it says
Contents of a sold product are not always what the label states. Nothing in this record points to this reason.
No recorded reason
RNAWiki has nothing stored that would explain it. Nothing in this record points to this reason.

None of these is a reason to take more. Taking more is not a step this page ever suggests.

What it would be like to takeRead from sources, not yet reviewed

What taking it involves

The recorded facts about getting hold of it and using it. Nothing here is a suggestion about what you should do.

How it is supplied

Still being tested

Quoted from a source

Where this came from

Copied from a source RNAWiki stored, with the source named.

Read from the recorded approval status.

No source is stored against this line.

Form and route

Oral tablet, capsule or liquid, 25 mg once daily in every published trial

Source-linked record

Where this came from

A person wrote this into the record with the study named beside it. No reviewer has signed it off.

The form and route recorded on this substance.

No source is stored against this line.

Who oversees it

Professional supervision is normally required. It is a prescription or clinician-administered medicine where it is approved.

Quoted from a source

Where this came from

Copied from a source RNAWiki stored, with the source named.

The identity record classes it as investigational; no register records an approval.

No source is stored against this line.

What is in the pack

A once-daily oral dose.

Source-linked record

Where this came from

A person wrote this into the record with the study named beside it. No reviewer has signed it off.

Recorded notes on how this is sold. The rest of the recorded wording: The unusual consistency of the clinical programme — 25 mg in all four major trials — means the human dose-response is narrow but the exposure is well characterised at that dose, over durations up to two years. That is far more human data than any SARM on this site has.

No source is stored against this line.

Where it is registered

Regulatory records are listed in the technical disclosure at the foot of this page.

Counted from records

Where this came from

A count of rows RNAWiki holds. It describes our records, not your body.

Register entries are stored per jurisdiction and shown with their dates.

No source is stored against this line.

Why people stop

Not recorded.

Nothing recorded

Where this came from

RNAWiki holds nothing here and says so rather than guessing.

No record of why people stopped taking it is stored for this substance.

No source is stored against this line.

What it would be like to takeRead from sources, not yet reviewed

What can go wrong

Sorted by where each line came from. A regulator's label and a report somebody sent in are not the same kind of fact.

Worth asking a clinician aboutWritten into the record from the studies named on this page

Documented in trials: increased appetite subsiding over months, transient mild lower-extremity oedema, muscle pain, a small rise in fasting glucose with decreased insulin sensitivity, and a rise in cortisol. The hip fracture phase 2b was terminated early for a congestive heart failure signal and its authors describe the safety profile in that population as unfavourable. Fluid retention and insulin resistance are the expected consequences of sustained growth hormone elevation and are the reason growth hormone itself is not given to healthy people. One published case of hepatotoxicity resolving on withdrawal.

Nobody counted how many people took this and were fine, so this cannot be turned into a rate.

Where this came from

Over a long time. No completed tested study window is recorded, so long-term effects are not established by the registry record.

Groups the studies covered thinly. Which groups were under-represented in the studies has not been recorded for this substance. Pregnancy, breastfeeding, children, older adults, and people with liver or kidney conditions are commonly excluded from trials; nothing here says whether they were.

What is missing or unclearRead from sources, not yet reviewed

Does the exact form matter?

Evidence belongs to the exact thing that was tested. A different salt, a different mixture or a different preparation is a different question.

The form that was studied

Oral tablet, capsule or liquid, 25 mg once daily in every published trial

Source-linked record

Where this came from

A person wrote this into the record with the study named beside it. No reviewer has signed it off.

The form used in the studies cited on this page.

No source is stored against this line.

What is sold

A recorded note compares this form with the others that are sold. It is kept below, word for word.

A fixed RNAWiki sentence

Where this came from

Wording RNAWiki always uses, not a finding about this substance.

The recorded note, unchanged: The unusual consistency of the clinical programme — 25 mg in all four major trials — means the human dose-response is narrow but the exposure is well characterised at that dose, over durations up to two years. That is far more human data than any SARM on this site has.

No source is stored against this line.

What is recorded as being sold

  • 6 products list this as an active ingredient in the United States drug directory. 6 of them contain it and nothing else.

    FDA National Drug Code directory · 85207-063 · read 2026-08-29

  • They are sold as powder.

    FDA National Drug Code directory · 85207-063 · read 2026-08-29

  • 9 marketed supplement labels list this ingredient, classed as non-nutrient/non-botanical, other combinations and other.

    NIH Dietary Supplement Label Database · 172529 · read 2026-08-29

  • Those labels carry all other, no claim and structure/function claims. A claim of that kind is written by the manufacturer and is not assessed by any regulator, so its presence says nothing about whether it is true.

    NIH Dietary Supplement Label Database · 172529 · read 2026-08-29

Evidence carries across two names for one substance. It does not carry across a salt, a mirror form or a mixture.

What it would be like to takeRead from sources, not yet reviewed

What you could measure

This one is decided with a clinician, so this section holds questions to ask rather than a plan to run.

Self-experiment planning is not offered for a prescription or clinician-supervised medicine, investigational agent. The questions below are for a clinician or pharmacist.

Questions worth asking

  • Which of the trials of Ibutamoren studied people like me?
  • What was measured, and for how long?
  • Was the result a laboratory value or a health outcome?
  • What would we watch for, and when would we stop?

Tracking can show whether something changed for you. It cannot show what caused it.

RNAWiki records evidence. It does not say whether this substance is right for you.

What is missing or unclearRead from sources, not yet reviewed

Claims that go past the evidence

Things said about this substance that sound reasonable, and the exact step that is missing between the evidence and the claim.

Goes past the evidence

That raising IGF-1 produces functional benefit — tested in three populations across 912 randomised participants and supported in none

Why it sounds right. It follows from something real on this page: a body step, an animal result or a related finding.

The missing step. Nobody measured this in people for this use, or the study that did measure it did not show it.

What would change this

A study in people, for this exact use, measuring this exact thing, that a reviewer signs off.

RNAWiki has not yet published a reviewed conclusion for this use.

Goes past the evidence

That a 1.1 kg fat-free mass gain represents contractile tissue, when body cell mass was inferred from intracellular water and strength did not change

Why it sounds right. It follows from something real on this page: a body step, an animal result or a related finding.

The missing step. Nobody measured this in people for this use, or the study that did measure it did not show it.

What would change this

A study in people, for this exact use, measuring this exact thing, that a reviewer signs off.

RNAWiki has not yet published a reviewed conclusion for this use.

Goes past the evidence

That the appetite and sleep effects reported by users are established outcomes; appetite increase is documented in the trials as an adverse effect that subsided, and sleep architecture was not a reported endpoint in any of them

Why it sounds right. It follows from something real on this page: a body step, an animal result or a related finding.

The missing step. Nobody measured this in people for this use, or the study that did measure it did not show it.

What would change this

A study in people, for this exact use, measuring this exact thing, that a reviewer signs off.

RNAWiki has not yet published a reviewed conclusion for this use.

What is missing or unclearRead from sources, not yet reviewed

What nobody knows yet

Open questions, each with why it is open and what would close it.

How much did people take in the studies?

The sources RNAWiki checked hold nothing for this field.

Why it matters. A result belongs to an amount. Without the amount the result floats free.

What would answer it

A stored source that records it, and a reviewer to sign it off.

Last checked 2026-09-04. Searched: The sources listed in the receipts at the foot of this page.

How fast does the body clear it?

The sources RNAWiki checked hold nothing for this field.

Why it matters. Without this, nothing on this page can say how long anything lasts.

What would answer it

A stored source that records it, and a reviewer to sign it off.

Last checked 2026-09-04. Searched: The sources listed in the receipts at the foot of this page.

Missing populations

Which groups were under-represented in the studies has not been recorded.

Why it matters. Recorded by the evidence model as a gap on this record.

What would answer it

A study that measures it, and a reviewer to sign it off.

Last checked 2026-09-04. Searched: The sources listed in the receipts at the foot of this page.

Missing long-term data

No completed tested study window is recorded.

Why it matters. Recorded by the evidence model as a gap on this record.

What would answer it

A study that measures it, and a reviewer to sign it off.

Last checked 2026-09-04. Searched: The sources listed in the receipts at the foot of this page.

No reviewed conclusion

RNAWiki has not yet published a reviewed conclusion for this use.

Why it matters. Recorded by the evidence model as a gap on this record.

What would answer it

A study that measures it, and a reviewer to sign it off.

Last checked 2026-09-04. Searched: The sources listed in the receipts at the foot of this page.

Missing interaction studies

No interaction was found in the registers checked. Not finding one is not the same as showing there is none.

Why it matters. Recorded by the evidence model as a gap on this record.

What would answer it

A study that measures it, and a reviewer to sign it off.

Last checked 2026-09-04. Searched: The sources listed in the receipts at the foot of this page.

Formulation uncertainty

Several salts, forms or products of Ibutamoren are recorded. Results from one form may not transfer to another.

Why it matters. Recorded by the evidence model as a gap on this record.

What would answer it

A study that measures it, and a reviewer to sign it off.

Last checked 2026-09-04. Searched: The sources listed in the receipts at the foot of this page.

Mechanism not reviewed

No reviewed mechanism story exists for this substance.

Why it matters. Recorded by the evidence model as a gap on this record.

What would answer it

A study that measures it, and a reviewer to sign it off.

Last checked 2026-09-04. Searched: The sources listed in the receipts at the foot of this page.

What happened in peopleRead from sources, not yet reviewed

Check any of this yourself

Every line above traced back to the study it came from. This is the technical layer, and the vocabulary changes here.

Every line above can be traced to the study named beside it. Follow the link and read it.

Two years in 65 older adults: fat-free mass up 1.1 kg, strength unchanged
In plain words
In a two-year randomised trial, ibutamoren raised growth hormone and IGF-1 into the young-adult range and added 1.1 kg of fat-free mass while placebo lost 0.5 kg. It made no difference to strength or function.
What was measured
Fat-free mass and abdominal visceral fat at 1 year, confirmed at 2 years
Effect estimate
No reviewed effect estimate exists for this record.
Limits
See the technical detail.
From the source
Nass et al. ran a two-year, double-blind, randomised, placebo-controlled, modified-crossover trial in 65 healthy adults aged 60 to 81, at 25 mg orally once daily. Growth hormone and IGF-1 rose into the healthy young-adult range. Fat-free mass changed by +1.1 kg (95% CI 0.7 to 1.5) on drug against -0.5 kg (-1.1 to 0.2) on placebo, P < 0.001; body cell mass by intracellular water followed the same pattern (P = 0.021). Body weight rose 2.7 kg against 0.8 kg (P = 0.003), with the average increase in limb fat greater on drug (1.1 kg versus 0.24 kg, P = 0.001) and no significant difference in abdominal visceral fat or total fat mass. The paper states plainly that the increased fat-free mass did not result in changes in strength or function, and that the two-year exploratory analyses confirmed the one-year results.
Source
Nass R et al., Ann Intern Med 2008;149:601-611
Role in the trial
Not matched to a registered study
Identity check
no open identity hold
Audit mark
verified
Review state
Written into the record, not signed off as a reviewed claim
Metabolic cost measured in the same trial: glucose up, insulin sensitivity down
In plain words
The same two-year trial recorded a rise in fasting blood sugar, a fall in insulin sensitivity and a rise in cortisol, alongside the muscle gain.
What was measured
Fasting glucose, insulin sensitivity, cortisol and lipid changes over 12 months
Effect estimate
No reviewed effect estimate exists for this record.
Limits
See the technical detail.
From the source
In Nass et al., fasting blood glucose rose an average of 0.3 mmol/L (5 mg/dL) on MK-677 (P = 0.015) and insulin sensitivity decreased. Cortisol rose 47 nmol/L (95% CI 28 to 71), equivalent to 1.7 micrograms/dL (P = 0.020). LDL cholesterol fell 0.14 mmol/L relative to baseline (P = 0.026) with no between-group difference in total or HDL cholesterol. The commonest side effects were an increase in appetite that subsided over months, and transient mild lower-extremity oedema and muscle pain. These are the mechanistic consequences of chronic growth hormone elevation and they are reported in the same paper as the fat-free mass gain; secondary accounts of this compound often carry the first result and not the second.
Source
Nass R et al., Ann Intern Med 2008;149:601-611
Role in the trial
Not matched to a registered study
Identity check
no open identity hold
Audit mark
verified
Review state
Written into the record, not signed off as a reviewed claim
Alzheimer disease: IGF-1 up 73%, and no effect on any clinical scale
In plain words
In 563 patients with Alzheimer disease, a year of ibutamoren raised IGF-1 by nearly three quarters and changed nothing about the disease on any of the four measures used.
What was measured
CIBIC-plus, ADAS-Cog, ADCS-ADL and CDR-sob at 12 months
Effect estimate
No reviewed effect estimate exists for this record.
Limits
This entry records a failure.
From the source
Sevigny et al. randomised 563 patients with mild to moderate Alzheimer disease to MK-677 25 mg or placebo daily for 12 months, with 416 completing. Serum IGF-1 rose 60.1% at six weeks and 72.9% at twelve months. In mixed-effects models there were no significant differences between groups on the CIBIC-plus, or on mean change from baseline in ADAS-Cog, ADCS-ADL or CDR sum of boxes. The authors state the conclusion in the paper title: despite evidence of target engagement, the drug was ineffective. This is the textbook shape of a target-engagement-without-clinical-benefit result, and it is a stronger negative than a trial that simply failed to reach its dose.
Source
Sevigny JJ et al., Neurology 2008;71:1702-1708
Role in the trial
Not matched to a registered study
Identity check
no open identity hold
Audit mark
verified
Review state
Written into the record, not signed off as a reviewed claim
A hip fracture trial stopped early for congestive heart failure
In plain words
A 123-patient trial in people recovering from a hip fracture was terminated before it finished, because of a heart failure safety signal in a small number of patients.
What was measured
Functional performance measures and IGF-1 at 24 weeks; reason for early termination
Effect estimate
No reviewed effect estimate exists for this record.
Limits
This entry records a failure.
From the source
Adunsky et al. randomised 123 elderly hip fracture patients to MK-0677 25 mg daily or placebo. IGF-1 rose 51.4 ng/mL against placebo (95% CI 34.42 to 68.44, P < 0.001). Mean stair climbing power at 24 weeks rose 12.5 W against placebo but the confidence interval crossed zero (95% CI -10.95 to 35.88, P = 0.292); gait speed showed a 0.7-score difference in means (95% CI 0.17 to 1.28, P = 0.011); several other functional measures showed no improvement. The trial was terminated early due to a safety signal of congestive heart failure in a limited number of patients, and the authors conclude that MK-0677 has an unfavourable safety profile in this population. An earlier 161-patient trial by Bach et al. had already found an 84% IGF-1 rise with no significant difference from placebo in functional performance or in the overall Sickness Impact Profile score.
Source
Adunsky A et al., Arch Gerontol Geriatr 2011;53:183-189; earlier trial Bach MA et al., J Am Geriatr Soc 2004;52:516-523
Role in the trial
Not matched to a registered study
Identity check
no open identity hold
Audit mark
verified
Review state
Written into the record, not signed off as a reviewed claim
IGF-1 is a target-engagement marker, not an outcome
In plain words
Every trial showed IGF-1 rising, which proves the drug reached its target. Not one trial showed that reaching the target helped anybody.
What was measured
That raising IGF-1 delivers the benefits attributed to growth hormone — the specific inference four randomised trials were built to test, and none supported
Effect estimate
No reviewed effect estimate exists for this record.
Limits
See the technical detail.
From the source
IGF-1 rose 84% in Bach et al., 51.4 ng/mL over placebo in Adunsky et al., 60.1% at six weeks and 72.9% at twelve months in Sevigny et al., and into the young-adult range in Nass et al. Across those four trials, spanning 912 randomised participants and three distinct populations, the clinical outcomes were: no change in strength or function in healthy older adults, no significant difference in functional performance after hip fracture in the larger of the two fracture trials, and no effect on any Alzheimer scale. A biomarker that moves in every trial while the outcome moves in none is the definition of an unvalidated surrogate, and the entire consumer case for this compound rests on that biomarker.
Source
Nass R et al., Ann Intern Med 2008;149:601-611; Sevigny JJ et al., Neurology 2008;71:1702-1708; Adunsky A et al., Arch Gerontol Geriatr 2011;53:183-189; Bach MA et al., J Am Geriatr Soc 2004;52:516-523
Role in the trial
Not matched to a registered study
Identity check
no open identity hold
Audit mark
caution
Review state
Written into the record, not signed off as a reviewed claim
Present as an undeclared drug in products, and hepatotoxicity reported
In plain words
Ibutamoren turned up inside products sold as SARMs that did not list it, and a case of liver injury in a man in his early thirties has been published.
What was measured
Frequency as an undeclared product ingredient; published hepatotoxicity case
Effect estimate
No reviewed effect estimate exists for this record.
Limits
See the technical detail.
From the source
In Van Wagoner et al., 17 of 44 internet-purchased products (39%) contained an unapproved drug other than a SARM — ibutamoren, GW501516 or SR9009. Cobani et al. report transaminitis in an otherwise healthy man in his early thirties after two months of MK-677, resolving to normal after stopping. Reversible gynaecomastia and hypogonadism have been reported after commercial performance-enhancing supplement use involving this class. As with every case report on these pages, this establishes that the event occurs and gives no rate.
Source
Van Wagoner RM et al., JAMA 2017;318:2004-2010; Cobani E et al., BMJ Case Rep 2025;18:e265728
Role in the trial
Not matched to a registered study
Identity check
no open identity hold
Audit mark
verified
Review state
Written into the record, not signed off as a reviewed claim

Where else this substance is registered

FDA substance identifier (UNII)
GJ0EGN38UL
CAS registry number
159634-47-6
PubChem compound
178024
ChEMBL
CHEMBL13817
WHO international nonproprietary name list entry
7682
RxNorm concept
1805438
EMA substance identifier
100000083649

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What is missing or unclearRead from sources, not yet reviewed

How this medicine reached us

Kept near the foot of the page. A historical event moves up only when it changes something about this substance today.

What the approval register records

  • The earliest marketing start date recorded for a listed product is 20191030.

    FDA National Drug Code directory · 85207-063 · read 2026-08-29

What is missing or unclearRead from sources, not yet reviewed

What to learn next

Ordered by what would most change how you read this page, starting with what is easiest to misunderstand.

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6 questions this page could not answer

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The older medicine-wide conclusion held in this record

Written for a clinical reader, and about the medicine as a whole rather than about one indication, population and set of trials. RNAWiki does not treat it as a programme conclusion and no reviewer has signed it off in that form. It is here word for word because it is what the record holds.

A growth hormone secretagogue that reliably does exactly what it says — IGF-1 up by 51 to 84 percent, fat-free mass up 1.1 kg against a 0.5 kg loss on placebo — and produced no measurable gain in strength, function or cognition in any trial that looked for one, before a hip fracture trial was stopped early for heart failure.

Recorded evidence blocks (7)

What did Ibutamoren's largest trial (64 people) and its longest (3.7 years) measure?


64 people in Ibutamoren's largest registered study, 3.7 years in its longest registered window, measuring MK-0677 25 mg is superior to placebo in reducing symptoms of fibromyalgia, as assessed by the Fibromyalgia Impact Questionnaire (FIQ) over a 24-week treatment period. ClinicalTrials.gov · 2026-09-01

3 phase2, 1 na; NCT00395291; 2010-05; no ageing endpoint recorded. Last human test completed 2024, NCT05364684.

Interpretation These counts include studies where Ibutamoren was a comparison treatment, a background treatment or an exposure in an observational study, and the longest window may be a planned end date. Trial roles have not yet been classified for this record.

Show the evidence
  • phase2
    3
  • na
    1
  • Last recorded human test NCT05364684
    2024-12-23

recorded 2026-09-01 · last checked 2026-09-04

Ibutamoren was tested only in human — what did it show?


human: mechanism-only (4): the rungs where Ibutamoren has a recorded finding. Europe PMC and ClinicalTrials.gov · organism ladder · 2026-09-01

MK-0677 25 mg is superior to placebo in reducing symptoms of fibromyalgia, as assessed by the Fibromyalgia Impact Questionnaire (FIQ) over a 24-week treatment… — the recorded outcome words.

Yeast C. elegans Drosophila Mouse Rat Dog Non-human primate Human mechanism-only
Show the evidence
  • human NCT00116129
    mechanism-only; MK-0677 25 mg is superior to placebo in reducing symptoms of fibromyalgia, as assessed by the Fibromyalgia Impact Questionnaire (FIQ) over a 24-week treatment period; 4

recorded 2026-09-01 · last checked 2026-09-04

Why did Ibutamoren's trial NCT01343641 stop?


1 recorded trial of Ibutamoren stopped. ClinicalTrials.gov · 2026-09-01

"Could not obtain drug supply from manufacturer"; 1 of 4 registered studies

Show the evidence
  • Trial NCT01343641
    withdrawn; "Could not obtain drug supply from manufacturer"

recorded 2026-09-01 · last checked 2026-09-04

Which one trial of Ibutamoren posted no result?


Posted no result
1 of 1 completed trials
Registrations
NCT00116129
Completion dates
oldest 2008-04
Show the evidence
  • Trial NCT00116129
    2008-04

At the median, Ibutamoren's trials enrolled 30.5 people — anything larger?


Median enrolment
30.5
Largest enrolment
64
Registered trials counted
4

Was Ibutamoren studied with caloric restriction?


caloric restriction is named in Ibutamoren's label sentences: "During the first week of caloric restriction (i.e. diet alone), daily nitrogen losses were similar for both treatment groups (mean +/- SE; MK-677 group -2.67 +/- 0.40 g/day vs. placebo group -2.83 +/- 0.26 g/day)." openfda-label+europepmc · 1998-02-01

1 recorded statement; caloric restriction

Show the evidence
  • caloric restriction
    During the first week of caloric restriction (i.e. diet alone), daily nitrogen losses were similar for both treatment groups (mean +/- SE; MK-677 group -2.67 +/- 0.40 g/day vs. placebo group -2.83 +/- 0.26 g/day).

recorded 1998-02-01 · last checked 2026-09-04

What is recorded about Ibutamoren and IGF-1?


"Ghrelin receptor agonist" — where Ibutamoren and IGF-1 appear together. chembl+open-targets+europepmc · CHEMBL13817 · 2026-09-04

IGF-1; PMID 19015485

Show the evidence

IGF-1

  • "Ghrelin receptor agonist"
  • PMID 19015485
    "Despite evidence of target engagement as indicated by an increase in serum insulin-like growth factor-1, the human growth hormone secretagogue MK-677 25 mg was ineffective at slowing the rate of progression of Alzheimer disease."

recorded 2026-09-04 · last checked 2026-09-04

Where it is registered
Identifiers, relations and other names

The exact record

ChEMBL id
CHEMBL13817
PubChem CID
178024
CAS number
159634-47-6
RxCUI
1805438
InChIKey
UMUPQWIGCOZEOY-JOCHJYFZSA-N
Also called
Ibutamoreno, IBUTAMOREN MESYLATE, Ibutamoren mesilate, growth hormone secretagogue mk-0677, IBUTAMOREN [NFLIS-DRUG], Ibutamoren [WHO-DD], ibutamoren [INN]
Development code
L-163,191, MK-0677, MK-677
Salt form
MK-677, also written MK-0677; ibutamoren mesylate
Sources (6)

Sources

ChEMBL 37 — CC BY-SA 3.0 Unported · ClinicalTrials.gov — US Government work · Europe PMC — metadata only, under the recorded legal gate · Open Targets 26.06 — CC0 · derived from this page's own recorded fields; no external licence · openFDA / DailyMed — US Government work

How these records are assembled · Which registers were checked

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  • source coverage passed: 6 source rows
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