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Etranacogene dezaparvovec

  • Gene therapy
  • Given by a clinician
  • Identity checked
  • Sources last checked 2026-09-04

This page shows what was measured, who it was measured in, and what that does not settle.

What Etranacogene dezaparvovec does in the body

One infusion, and for most patients the weekly injections stop.

Your liver is supposed to make a clotting protein and does not. A harmless virus shell carries a working copy of the gene into liver cells, where it stays as a separate loop of DNA and starts producing the protein. The copy delivered is not the ordinary version — it is a naturally occurring variant found in an Italian family, about eight times more active than normal, so even modest production gives a useful clotting level.

Why people take it. Haemophilia B in adults

What happened in people

Annualised bleeding rate 4.19 during prophylaxis lead-in versus 1.51 during months 7 to 18 after treatment, rate ratio 0.36 (P<0.001)

Source-linked record

Where this came from

A person wrote this into the record with the study named beside it. No reviewer has signed it off.

The recorded finding that sits closest to something a person would notice. Written into the record, not signed off, and it carries no population or interval.

The limit that matters most

That the effect is lifelong — five years is the measured horizon and hepatocytes do turn over

Where it acts
Liver hepatocyte nucleus
Kind of result
The kind of result is not recorded
Supervision
Professional supervision is normally required. It is a prescription or clinician-administered medicine where it is approved.

What the registries record it as

  • The substance registry classes this as structurallydiverse.

    FDA substance registry · Z5XCD5Q9RL · read 2026-08-29

  • The material is recorded as coming from recombinant virus.

    FDA substance registry · Z5XCD5Q9RL · read 2026-08-29

Where each sentence above came from

A person wrote this explanation into the record, with the studies named in the path below.

The recorded use, written for a reader without medical training. Not signed off.

A limit recorded against this substance. Not signed off as a reviewed claim.

The four opening statements run to 114 words.

Words this page uses

Four words worth knowing first

Chosen from what this page shows, with each one explained before the word it depends on.

Absolute difference

An absolute difference is how many more people in a hundred were affected.

A picture of it, and where the picture fails

It is like counting heads in two rooms of a hundred.

Where that stops being true. Heads are easy to count. Study results carry a margin of error too.

What people get wrong. It is confused with a percentage change, which can look far larger.

The arithmetic difference in event rates between arms.

Confidence interval

A confidence interval is the range the true answer is likely to sit in.

A picture of it, and where the picture fails

It is like a weather forecast giving a range rather than one number.

Where that stops being true. A forecast range covers tomorrow. This one covers what the study could resolve.

What people get wrong. The middle number is read as the answer. A range crossing zero means no change is still possible.

An interval estimate that would contain the true parameter in a stated proportion of repeated studies.

What happened in peopleRead from sources, not yet reviewed

What happened in people

Each card is one study: the exact question it asked, who was in it, and whether it showed what it set out to show.

Annualised bleeding rate during months 7 to 18 after treatment compared with the prophylaxis lead-in period, tested for non-inferiority against a margin of 1.8

The study showed what it set out to show

Who was studied
HOPE-B (NCT03569891)
How many people
54
Study design
Phase 3, open label, single group with prospective prophylaxis lead-in
Compared against
Not recorded for this study
Kind of result
A number that stands in for health
What was found
P < 0.001; rate ratio 0.36 (95% Wald CI 0.20 to 0.64)
Repeated elsewhere
Partially Replicated

What this does not prove. Meeting one endpoint in one population does not carry to other goals or other people.

The limits of this study, and where it came from

Main limit. No treatment-related serious adverse events in the primary analysis. One case of hepatocellular carcinoma in a participant with extensive independent risk factors triggered an FDA clinical hold in December 2020; independent integration analysis concluded the tumour was unrelated to treatment.

How far it carries. This study is recorded from the curated record, not from the registry match.

Form and route. Recombinant AAV5 vector, single intravenous infusion

Interval reported. Not recorded in this summary

Written into the record, not signed off as a reviewed claim.

Adjusted annualised bleeding rate months 7 to 60 versus lead-in, plus factor IX expression and safety

The study showed what it set out to show

Who was studied
HOPE-B five-year final analysis (NCT03569891)
How many people
54
Study design
Phase 3, prespecified five-year analysis
Compared against
Not recorded for this study
Kind of result
A number that stands in for health
What was found
63% reduction in annualised bleeding rate (95% CI 24 to 82)
Repeated elsewhere
Partially Replicated

What this does not prove. Meeting one endpoint in one population does not carry to other goals or other people.

The limits of this study, and where it came from

Main limit. Adverse events possibly related to treatment were rare after month 6. Long-term hepatic surveillance remains a class requirement rather than a resolved question.

How far it carries. This study is recorded from the curated record, not from the registry match.

Form and route. Recombinant AAV5 vector, single intravenous infusion

Interval reported. 95% CI 24 to 82)

Written into the record, not signed off as a reviewed claim.

What we know
RNAWiki holds 2 written-up human studies for this substance, each with the question it asked.
How we know it
Each card names the study, the number of people and what the study set out to measure.
What this does not prove
These summaries were written by a person and have not been signed off as reviewed claims.
Why it matters
A study that failed is as informative as one that worked, and both are here.
What we still do not know
No reviewed claim exists, so no exact population, effect size or interval is published yet.

What it changes in the bodyRead from sources, not yet reviewed

The path through the body

From what a person takes to what changes. A solid line is a step measured in people. A dashed line is a step nobody has measured that way.

  1. Start

    Etranacogene dezaparvovec

    What a person takes: Recombinant AAV5 vector, single intravenous infusion.

    The measurement behind this step

    One intravenous infusion of 2 x 10^13 genome copies per kg body weight, given in a single session with no conditioning, no surgery and no prophylactic immunosuppression. Corticosteroid is reserved for reactive management of transaminase elevation.

  2. Getting in

    A single intravenous infusion

    One infusion into a vein. No chemotherapy, no surgery, no cells taken out of the body.

    Described, with no measurement named. No measurement is named for this step, so it is drawn as an unconfirmed link.

    The measurement behind this step

    A single dose of 2 x 10^13 genome copies per kg of AAV5 vector is infused intravenously. Corticosteroid is not given prophylactically; it is started reactively if transaminases rise.

  3. Reaching the cell

    The AAV5 shell is taken up by liver cells

    The blood carries the whole dose through the liver, where the shell is absorbed by the cells that make clotting proteins.

    Described, with no measurement named. No measurement is named for this step, so it is drawn as an unconfirmed link.

    The measurement behind this step

    AAV5 has strong natural hepatotropism after systemic administration. Uptake is receptor-mediated and the vast majority of the dose is cleared by the liver on first pass, which is why a liver-expressed protein is the natural target for systemic AAV.

  4. What it acts on

    A liver-only promoter keeps expression where it belongs

    The delivered gene comes with a switch that only liver cells can read, so it does not start producing clotting factor anywhere else.

    Described, with no measurement named. No measurement is named for this step, so it is drawn as an unconfirmed link.

    The measurement behind this step

    The codon-optimised factor IX Padua cassette is driven by the liver-specific LP1 promoter and persists as a non-integrating nuclear episome. Restricting expression to hepatocytes limits both off-target production and the immune presentation of the transgene product elsewhere.

  5. The change it makes

    The gene delivered is a hyperactive natural variant

    The copy inserted is not the standard one. It is a variant found in an Italian family whose members clot about eight times more efficiently per molecule, so a little protein goes a long way.

    Described, with no measurement named. No measurement is named for this step, so it is drawn as an unconfirmed link.

    The measurement behind this step

    Factor IX Padua carries an arginine-to-leucine substitution at position 338, first identified in a family with unexplained thrombophilia, conferring roughly eight-fold higher specific activity. This is the single design decision that made haemophilia B gene therapy clinically useful: it converts a modest, achievable expression level into a therapeutic clotting activity.

  6. What that does for a person

    Bleeding falls, and stays fallen for five years

    Bleeds dropped by about two thirds, weekly infusions largely stopped, and five years on the clotting factor level was still around a third of normal.

    Measured in people. Written by a person from the study named beside the step. Not signed off as a reviewed claim.

    The measurement behind this step

    Annualised bleeding rate 4.19 to 1.51 in the primary analysis (rate ratio 0.36); at five years, adjusted annualised bleeding rate 4.16 to 1.52, mean factor IX activity 36.1 ± 15.7 IU/dL, and a 96% reduction in exogenous factor IX consumption. Post-mitotic hepatocyte turnover in adults is slow enough that episomal loss did not materially erode expression over that horizon.

No suggested links are held for this record, so nothing is hidden from this path.

What we know
The record describes 5 steps from taking it to an effect in a person.
How we know it
Each step names the study behind it in the technical detail beside it.
What this does not prove
A change inside the body is a reason to look. It is not a result in a person.
Why it matters
A reader can see exactly where the chain stops being measured in people.
What we still do not know
No reviewed claim binds any of these steps to a named population.

What is missing or unclearRead from sources, not yet reviewed

Were people like you studied?

RNAWiki cannot tell whether a study fits you. It can show who was in it, and where the result stops carrying.

Who was studied

People similar to this profile were included.

  • Adults on factor IX prophylaxis, or with a history of life-threatening or repeated serious bleeding. Unusually for this field, patients were enrolled regardless of pre-existing antibodies to the AAV5 capsid.

Who is missing from the studies

  • Which groups were under-represented in the studies has not been recorded for this substance. Pregnancy, breastfeeding, children, older adults, and people with liver or kidney conditions are commonly excluded from trials; nothing here says whether they were.

What the label states about particular groups

  • On pediatric, the label states: “The safety and efficacy of HEMGENIX in pediatric patients have not been established.”

    US prescribing information · 35b2db65-4c6c-4173-ab56-b2bca69193bd · read 2026-08-30

  • On older people, the label states: “The clinical studies included a total of 6 geriatric patients with Hemophilia B, aged 68 to 75 years at time of enrollment.”

    US prescribing information · 35b2db65-4c6c-4173-ab56-b2bca69193bd · read 2026-08-30

  • On people who are pregnant, the label states: “Risk Summary HEMGENIX is not intended for administration in women.”

    US prescribing information · 35b2db65-4c6c-4173-ab56-b2bca69193bd · read 2026-08-30

  • On people with reduced liver function, the label states: “Limited clinical data in patients with liver impairment indicate numerically lower FIX activity as compared to patients without hepatic impairment [see Clinical Pharmacology (12.3) ] .”

    US prescribing information · 35b2db65-4c6c-4173-ab56-b2bca69193bd · read 2026-08-30

  • On people with reduced kidney function, the label states: “Limited clinical data are available in patients with mild and moderate renal impairment [see Clinical Pharmacology (12.3) ] .”

    US prescribing information · 35b2db65-4c6c-4173-ab56-b2bca69193bd · read 2026-08-30

Where the result stopped carrying

  • An FDA clinical hold in December 2020 after a hepatocellular carcinoma case, resolved only after independent vector integration and whole genome analysis
  • This is a scope explorer, not a diagnosis engine.
  • It shows who was studied so you can see whether people similar to you were included.

RNAWiki cannot tell whether a study fits you. It can show who was in it.

What it would be like to takeRead from sources, not yet reviewed

Why it might seem to do nothing

A real effect and a noticed effect are different things. These are the recorded reasons the two come apart.

It was studied in different people

The people in the studies were not much like the reader.

On this record: Which groups were under-represented in the studies has not been recorded for this substance. Pregnancy, breastfeeding, children, older adults, and people with liver or kidney conditions are commonly excluded from trials; nothing here says whether they were.

The evidence may simply be wrong

Small early studies often do not hold up.

On this record: RNAWiki has not yet published a reviewed conclusion for this use.

Other reasons RNAWiki checked and found nothing for (11)
It was studied for a different goal
The studies measured something else entirely. Nothing in this record points to this reason.
A different form was studied
The studied form is not the form on the shelf. Nothing in this record points to this reason.
There was nothing to correct
Where a level is already normal, topping it up may change nothing. Nothing in this record points to this reason.
Something else had to happen too
In the studies it was paired with training, a diet or another treatment. Nothing in this record points to this reason.
The change is too small to feel
A real change can still sit below what a person notices. Nothing in this record points to this reason.
Day-to-day swing hides it
Sleep, food, stress and time of day move most numbers more than this would. Nothing in this record points to this reason.
Not enough time yet
The studies ran longer than the person has waited. Nothing in this record points to this reason.
It was not taken as studied
Missed days change the result, and studies count them. Nothing in this record points to this reason.
Something else changed at the same time
Two changes at once cannot be told apart afterwards. Nothing in this record points to this reason.
The product may not be what it says
Contents of a sold product are not always what the label states. Nothing in this record points to this reason.
No recorded reason
RNAWiki has nothing stored that would explain it. Nothing in this record points to this reason.

None of these is a reason to take more. Taking more is not a step this page ever suggests.

What it would be like to takeRead from sources, not yet reviewed

What taking it involves

The recorded facts about getting hold of it and using it. Nothing here is a suggestion about what you should do.

How it is supplied

Given by a clinician

Quoted from a source

Where this came from

Copied from a source RNAWiki stored, with the source named.

Read from the recorded approval status.

No source is stored against this line.

Form and route

Recombinant AAV5 vector, single intravenous infusion

Source-linked record

Where this came from

A person wrote this into the record with the study named beside it. No reviewer has signed it off.

The form and route recorded on this substance.

No source is stored against this line.

Who oversees it

Professional supervision is normally required. It is a prescription or clinician-administered medicine where it is approved.

Quoted from a source

Where this came from

Copied from a source RNAWiki stored, with the source named.

The identity record classes it as CRISPR / Gene Therapy.

No source is stored against this line.

What is in the pack

One intravenous infusion of 2 x 10^13 genome copies per kg body weight, given in a single session with no conditioning, no surgery and no prophylactic immunosuppression. Corticosteroid is reserved for reactive management of transaminase elevation.

Source-linked record

Where this came from

A person wrote this into the record with the study named beside it. No reviewer has signed it off.

Recorded notes on how this is sold and what that means for what is in the pack.

No source is stored against this line.

Where it is registered

Regulatory records are listed in the technical disclosure at the foot of this page.

Counted from records

Where this came from

A count of rows RNAWiki holds. It describes our records, not your body.

Register entries are stored per jurisdiction and shown with their dates.

No source is stored against this line.

Why people stop

Not recorded.

Nothing recorded

Where this came from

RNAWiki holds nothing here and says so rather than guessing.

No record of why people stopped taking it is stored for this substance.

No source is stored against this line.

What it would be like to takeRead from sources, not yet reviewed

What can go wrong

Sorted by where each line came from. A regulator's label and a report somebody sent in are not the same kind of fact.

Worth asking a clinician aboutWritten into the record from the studies named on this page

No boxed warning. The principal labelled concerns are infusion reactions, hepatotoxicity with transaminase elevation requiring monitoring and possible corticosteroid treatment, and the theoretical risk of hepatocellular carcinoma warranting long-term liver surveillance particularly in patients with cirrhosis, advanced fibrosis or chronic hepatitis. Factor IX inhibitor development was not observed in the pivotal programme.

Nobody counted how many people took this and were fine, so this cannot be turned into a rate.

Where this came from

Over a long time. No completed tested study window is recorded, so long-term effects are not established by the registry record.

Groups the studies covered thinly. Which groups were under-represented in the studies has not been recorded for this substance. Pregnancy, breastfeeding, children, older adults, and people with liver or kidney conditions are commonly excluded from trials; nothing here says whether they were.

What is missing or unclearRead from sources, not yet reviewed

Does the exact form matter?

Evidence belongs to the exact thing that was tested. A different salt, a different mixture or a different preparation is a different question.

The form that was studied

Recombinant AAV5 vector, single intravenous infusion

Source-linked record

Where this came from

A person wrote this into the record with the study named beside it. No reviewer has signed it off.

The form used in the studies cited on this page.

No source is stored against this line.

What is sold

Corticosteroid is reserved for reactive management of transaminase elevation.

Source-linked record

Where this came from

A person wrote this into the record with the study named beside it. No reviewer has signed it off.

Recorded notes on which forms are sold and how they compare.

No source is stored against this line.

What is recorded as being sold

  • 1 published label names it as an active ingredient. 1 of them describe this substance alone, which is where its own label text on this page comes from.

    US prescribing information · 35b2db65-4c6c-4173-ab56-b2bca69193bd · read 2026-08-29

  • Those labels are classed as human prescription drug.

    US prescribing information · 35b2db65-4c6c-4173-ab56-b2bca69193bd · read 2026-08-29

Evidence carries across two names for one substance. It does not carry across a salt, a mirror form or a mixture.

What it would be like to takeRead from sources, not yet reviewed

What you could measure

This one is decided with a clinician, so this section holds questions to ask rather than a plan to run.

Self-experiment planning is not offered for a prescription or clinician-supervised medicine. The questions below are for a clinician or pharmacist.

Questions worth asking

  • Which of the trials of Etranacogene dezaparvovec studied people like me?
  • What was measured, and for how long?
  • Was the result a laboratory value or a health outcome?
  • What would we watch for, and when would we stop?

Tracking can show whether something changed for you. It cannot show what caused it.

RNAWiki records evidence. It does not say whether this substance is right for you.

What is missing or unclearRead from sources, not yet reviewed

Claims that go past the evidence

Things said about this substance that sound reasonable, and the exact step that is missing between the evidence and the claim.

Goes past the evidence

That the effect is lifelong — five years is the measured horizon and hepatocytes do turn over

Why it sounds right. It follows from something real on this page: a body step, an animal result or a related finding.

The missing step. Nobody measured this in people for this use, or the study that did measure it did not show it.

What would change this

A study in people, for this exact use, measuring this exact thing, that a reviewer signs off.

RNAWiki has not yet published a reviewed conclusion for this use.

Goes past the evidence

That the unblinded within-patient bleeding comparison is equivalent to a randomised blinded one

Why it sounds right. It follows from something real on this page: a body step, an animal result or a related finding.

The missing step. Nobody measured this in people for this use, or the study that did measure it did not show it.

What would change this

A study in people, for this exact use, measuring this exact thing, that a reviewer signs off.

RNAWiki has not yet published a reviewed conclusion for this use.

Goes past the evidence

That the $3.5 million price is offset, which requires projecting factor IX savings decades beyond the measured five years

Why it sounds right. It follows from something real on this page: a body step, an animal result or a related finding.

The missing step. Nobody measured this in people for this use, or the study that did measure it did not show it.

What would change this

A study in people, for this exact use, measuring this exact thing, that a reviewer signs off.

RNAWiki has not yet published a reviewed conclusion for this use.

Goes past the evidence

That the hepatocellular carcinoma question is closed for the class; low-rate AAV integration keeps hepatic surveillance a standing requirement

Why it sounds right. It follows from something real on this page: a body step, an animal result or a related finding.

The missing step. Nobody measured this in people for this use, or the study that did measure it did not show it.

What would change this

A study in people, for this exact use, measuring this exact thing, that a reviewer signs off.

RNAWiki has not yet published a reviewed conclusion for this use.

What is missing or unclearRead from sources, not yet reviewed

What nobody knows yet

Open questions, each with why it is open and what would close it.

How much did people take in the studies?

The sources RNAWiki checked hold nothing for this field.

Why it matters. A result belongs to an amount. Without the amount the result floats free.

What would answer it

A stored source that records it, and a reviewer to sign it off.

Last checked 2026-09-04. Searched: The sources listed in the receipts at the foot of this page.

Missing populations

Which groups were under-represented in the studies has not been recorded.

Why it matters. Recorded by the evidence model as a gap on this record.

What would answer it

A study that measures it, and a reviewer to sign it off.

Last checked 2026-09-04. Searched: The sources listed in the receipts at the foot of this page.

Missing long-term data

No completed tested study window is recorded.

Why it matters. Recorded by the evidence model as a gap on this record.

What would answer it

A study that measures it, and a reviewer to sign it off.

Last checked 2026-09-04. Searched: The sources listed in the receipts at the foot of this page.

No reviewed conclusion

RNAWiki has not yet published a reviewed conclusion for this use.

Why it matters. Recorded by the evidence model as a gap on this record.

What would answer it

A study that measures it, and a reviewer to sign it off.

Last checked 2026-09-04. Searched: The sources listed in the receipts at the foot of this page.

Missing interaction studies

No interaction was found in the registers checked. Not finding one is not the same as showing there is none.

Why it matters. Recorded by the evidence model as a gap on this record.

What would answer it

A study that measures it, and a reviewer to sign it off.

Last checked 2026-09-04. Searched: The sources listed in the receipts at the foot of this page.

Mechanism not reviewed

No reviewed mechanism story exists for this substance.

Why it matters. Recorded by the evidence model as a gap on this record.

What would answer it

A study that measures it, and a reviewer to sign it off.

Last checked 2026-09-04. Searched: The sources listed in the receipts at the foot of this page.

What happened in peopleRead from sources, not yet reviewed

Check any of this yourself

Every line above traced back to the study it came from. This is the technical layer, and the vocabulary changes here.

Every line above can be traced to the study named beside it. Follow the link and read it.

HOPE-B: annualised bleeding rate fell 64% against each patient's own prophylaxis
In plain words
Fifty-four men spent at least six months on standard prophylaxis first, so their own bleeding rate was measured before treatment. After one infusion, bleeds fell by roughly two thirds.
What was measured
Annualised bleeding rate 4.19 during lead-in versus 1.51 after treatment; rate ratio 0.36
Effect estimate
No reviewed effect estimate exists for this record.
Limits
See the technical detail.
From the source
Open-label phase 3 study with a lead-in period of at least six months of factor IX prophylaxis, then a single 2 x 10^13 gc/kg infusion in 54 men with factor IX activity 2% or less, enrolled regardless of pre-existing AAV5 neutralising antibodies. The annualised bleeding rate fell from 4.19 (95% CI 3.22 to 5.45) during lead-in to 1.51 (95% CI 0.81 to 2.82) during months 7 through 18, a rate ratio of 0.36 (95% Wald CI 0.20 to 0.64; P<0.001), establishing both non-inferiority against the 1.8 margin and superiority. Factor IX activity rose by a least-squares mean of 36.2 percentage points at 6 months and 34.3 at 18 months, and factor IX concentrate use fell by a mean 248,825 IU per participant per year (P<0.001 for all three). No treatment-related serious adverse events occurred.
Source
Pipe SW et al., N Engl J Med 2023;388:706-718 (HOPE-B)
Role in the trial
Not matched to a registered study
Identity check
no open identity hold
Audit mark
verified
Review state
Written into the record, not signed off as a reviewed claim
Five-year factor IX activity of 36.1 IU/dL — durability that did not decay
In plain words
Five years after a single infusion, average clotting factor levels were essentially where they had been at eighteen months. That is the result this whole field has been trying to produce.
What was measured
Mean factor IX activity at 5 years: 36.1 ± 15.7 IU/dL
Effect estimate
No reviewed effect estimate exists for this record.
Limits
See the technical detail.
From the source
Prespecified five-year analysis of all 54 participants. The adjusted annualised bleeding rate for all bleeds was 4.16 during lead-in and 1.52 during months 7 through 60, a 63% reduction (95% CI 24 to 82). Mean factor IX activity at five years was 36.1 ± 15.7 IU per deciliter. Mean exogenous factor IX consumption fell 96%, from 257,339 IU per year in lead-in to 10,924 IU per year post-treatment. Efficacy did not differ substantially between participants with and without baseline AAV5 neutralising antibodies. Treatment-related adverse events were rare after month 6.
Source
Pipe SW et al., N Engl J Med 2025 (HOPE-B final analysis)
Role in the trial
Not matched to a registered study
Identity check
no open identity hold
Audit mark
verified
Review state
Written into the record, not signed off as a reviewed claim
Pre-existing AAV5 antibodies were expected to disqualify patients, and mostly did not
In plain words
Most gene therapies exclude anyone whose immune system has already met the virus shell. This trial enrolled them anyway, and the treatment still worked below a certain antibody level.
What was measured
Not recorded
Effect estimate
No reviewed effect estimate exists for this record.
Limits
See the technical detail.
From the source
HOPE-B enrolled participants regardless of pre-existing AAV5 neutralising antibody status, a deliberate departure from the field's standard exclusion. Benefit and safety were observed in participants with predose titres below 700, and the five-year analysis found efficacy did not differ substantially between antibody-positive and antibody-negative participants. The contrast with the competing haemophilia B product is stark: in BENEGENE-2, 188 of 316 men screened (59.5%) were ineligible because of anti-AAV neutralising antibodies. Capsid choice and screening policy, not the transgene, decide who is treatable.
Source
Pipe 2023 and the HOPE-B five-year analysis; contrast with Cuker A et al., N Engl J Med 2024;391:1108-1118
Role in the trial
Not matched to a registered study
Identity check
no open identity hold
Audit mark
verified
Review state
Written into the record, not signed off as a reviewed claim
A liver cancer case put the whole programme on FDA clinical hold in December 2020
In plain words
One participant developed liver cancer and the FDA halted the programme. A full genetic investigation concluded the therapy was unlikely to have caused it — but the hold happened, and the question it raised has not gone away for the field.
What was measured
Not recorded
Effect estimate
No reviewed effect estimate exists for this record.
Limits
This entry records a failure.
From the source
In December 2020 the FDA placed a clinical hold on the programme after a serious adverse event of hepatocellular carcinoma in a HOPE-B participant. The patient had multiple independent risk factors: prior hepatitis B and C, evidence of non-alcoholic fatty liver disease, a smoking history, a family history of cancer and advanced age. Independent molecular characterisation and vector integration analysis of the tumour and adjacent tissue supported the conclusion that the carcinoma was unrelated to treatment, and whole genome sequencing showed chromosome 1 and 8 abnormalities characteristic of hepatocellular carcinoma along with TP53 and other oncogenic mutations. The hold was lifted and the programme completed. AAV genomes are predominantly episomal but integrate at a low rate, so hepatic malignancy surveillance after liver-directed AAV is a permanent feature of this class rather than a resolved question.
Source
ASH Clinical News, reporting the independent investigation of the HOPE-B hepatocellular carcinoma case
Role in the trial
Not matched to a registered study
Identity check
no open identity hold
Audit mark
caution
Review state
Written into the record, not signed off as a reviewed claim
The comparator was each patient's own unblinded lead-in, not a randomised arm
In plain words
There was no control group. Every participant was compared with their own bleeding rate from the months before treatment, and everyone knew who had been treated.
What was measured
That an unblinded within-patient bleeding-rate comparison carries the same weight as a randomised blinded one
Effect estimate
No reviewed effect estimate exists for this record.
Limits
See the technical detail.
From the source
HOPE-B is open-label and single-group. The lead-in design is a genuine strength — it establishes each patient's own prophylaxis-era bleeding rate prospectively rather than by recall — but it cannot control for regression to the mean, for the behavioural effect of knowing one has been treated, or for changes in bleed reporting once weekly infusions stop. Annualised bleeding rate is patient-reported and adjudicated, not instrumented. Factor IX activity, by contrast, is a laboratory measurement and is not vulnerable to any of this.
Source
HOPE-B study design (NCT03569891)
Role in the trial
Not matched to a registered study
Identity check
no open identity hold
Audit mark
caution
Review state
Written into the record, not signed off as a reviewed claim
A $3.5 million price justified by lifetime factor costs that were never measured against it
In plain words
The price was set on the argument that a lifetime of clotting factor costs more. The trial measured five years, and the arithmetic beyond that is a projection.
What was measured
That five years of measured factor IX savings establishes lifetime cost offset at the launch price
Effect estimate
No reviewed effect estimate exists for this record.
Limits
See the technical detail.
From the source
CSL Behring set a US list price of $3.5 million on approval in November 2022, making it the most expensive medicine in the world at the time. The offsetting-cost argument rests on measured factor IX consumption — 257,339 IU per participant per year during lead-in, falling 96% to 10,924 IU — extrapolated across a lifetime. The measured horizon is five years. No peer-reviewed cost of goods for the product exists, so no markup can be stated, and this page states none.
Source
Fierce Pharma, 22 November 2022 (list price); HOPE-B five-year factor IX consumption data
Role in the trial
Not matched to a registered study
Identity check
no open identity hold
Audit mark
caution
Review state
Written into the record, not signed off as a reviewed claim

Where else this substance is registered

FDA substance identifier (UNII)
Z5XCD5Q9RL
CAS registry number
2156583-26-3
WHO international nonproprietary name list entry
10963
EMA substance identifier
300000005896

Checks this page had to pass

  • Passed

    Identity resolved

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  • Passed

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  • Passed

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  • Not passed

    Trial roles classified for highlighted evidence

    No registered study is classified as testing this substance.

  • Passed

    No internal keys in reader text

    enforced by the copy-contract test over the rendered page

  • Passed

    Safety mode resolved

    The identity record classes it as CRISPR / Gene Therapy.

  • Passed

    Canonical metadata present

    slug and display name present

What is missing or unclearRead from sources, not yet reviewed

What to learn next

Ordered by what would most change how you read this page, starting with what is easiest to misunderstand.

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What is not here

8 questions this page could not answer

These sections were prepared and left out because there was nothing to put in them. They are listed rather than hidden, so the gaps in this record are visible.

  • What was measured, goal by goal — found nothing in the sources checked.
  • How close this is to real life — found nothing in the sources checked.
  • Felt, measured, or meaningful — found nothing in the sources checked.
  • How long anything takes — found nothing in the sources checked.
  • What it may clash with — found nothing in the sources checked.
  • Other ways to the same goal — found nothing in the sources checked.
  • How this medicine reached us — found nothing in the sources checked.
  • What changed on this page — found nothing in the sources checked.

The record as stored

The full record, for auditing

Everything above is built from what is below. This layer keeps the technical vocabulary, record identifiers and every stored row, so a reader who wants to check the page can.

The older medicine-wide conclusion held in this record

Written for a clinical reader, and about the medicine as a whole rather than about one indication, population and set of trials. RNAWiki does not treat it as a programme conclusion and no reviewer has signed it off in that form. It is here word for word because it is what the record holds.

A single AAV5 infusion carrying the hyperactive factor IX Padua variant: the annualised bleeding rate fell 64% against each patient's own prophylaxis lead-in, and at five years mean factor IX activity was still 36.1 IU/dL — the strongest durability result any liver-directed gene therapy has published.

Recorded evidence blocks (3)

On the Etranacogene dezaparvovec label: indicated for what?


"HEMGENIX is indicated for treatment of adults with Hemophilia B (congenital Factor IX deficiency) who: Currently use Factor IX prophylaxis therapy, or Have current or historical life-threatening hemorrhage, or Have repeated, serious spontaneous bleeding episodes. HEMGENIX is an adeno-associated virus vector-based gene…": indications and usage on Etranacogene dezaparvovec's label. DailyMed label · 35b2db65-4c6c-4173-ab56-b2bca69193bd · 2026-05-01

6 registered trials of Etranacogene dezaparvovec — at which phases?


Registered studies posting no result
4 of 6

6 registered studies of Etranacogene dezaparvovec: 3 phase3, 2 na or unstated, 1 phase2. CLINICALTRIALS_SNAPSHOT · 2026-09-01

6 with a PubMed record

Show the evidence
  • phase3
    3
  • na or unstated
    2
  • phase2
    1
  • recruiting
    3
  • completed
    2
  • enrolling by invitation
    1

recorded 2026-09-01 · last checked 2026-09-04

At the median, Etranacogene dezaparvovec's trials enrolled 45.5 people — anything larger?


Median enrolment
45.5
Largest enrolment
500
Registered trials counted
6
Where it is registered
Identifiers, relations and other names

The exact record

ChEMBL id
CHEMBL5095451
CAS number
2156583-26-3
Development code
AAV5-hFIXco-Padua, AMT-061
Also called
Etranacogene dezaparvovec-dlrb, Hemgenix, ETRANACOGENE DEZAPARVOVEC [USAN], Etranacogene dezaparvovec [MI], Etranacogene dezaparvovec [WHO-DD], etranacogene dezaparvovec [INN]
Sources (4)

Sources

ChEMBL 37 — CC BY-SA 3.0 Unported · ChEMBL ATC CC BY-SA · ClinicalTrials.gov — US Government work · mixed register set; per-register licences in docs/specs/corpus-20k-sources.md · openFDA / DailyMed — US Government work

How these records are assembled · Which registers were checked

Index-quality checks
  • identity passed: no open identity hold
  • required summary fields resolved: 4 required field(s) not terminal: Why people use it, Best-supported result, Biggest unanswered question, Human evidence
  • public claims reviewed: 0 reviewed claim(s); drafts are never rendered
  • source coverage passed: 4 source rows
  • no critical contamination: no quarantine open
  • canonical metadata passed: slug and display name present
  • no raw internal fields: enforced by the copy-contract test over the rendered page

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